Traumatic Memory are memories that are characterized by fragmented and intense sensations and emotions, often with little or no episodic memory attached.
When the state of hyper-arousal and neurophysiological activation associated with a traumatic event persists, the brain changes (Perry 1988, Perry 1991). In much the same way that repeated exposure to a cognitive stimulus results in a cognitive memory, repeated or continuous exposure to this state of arousal results in a 'state memory'. The system has been in the aroused state so much that the brain, likely using the same molecular mechanisms underlying learning/memory (Kandel 1989), has readjusted to these new environmental demands. The arousal is no longer reversible; there is a new homeostatic set point for neurotransmitter systems (e.g., norepinephrine, epinephrine) in the brain which regulate arousal, attention, affect and sympathetic nervous system functioning (Perry 1991). These 'state memories' are the basis for the characteristic symptoms in PTSD. The hypervigilance, increased startle, affective lability, anxiety, dysphoria, and the increased SNS activity and reactivity can all be related to the altered functioning key neurotransmitter systems in the brain (see Krystal et al., 1989; Perry et al., 1990; Perry 1991).
- Traumatic Memory Inventory
- ↑ van der Kolk, B., Hopper, J., Osterman, J., (2001). Exploring the nature of traumatic memory, Journal of aggression, maltreatment and trauma, 4(2), 9-31.
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