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There is now significant evidence that [[prenatal]] exposure to infections increases the risk for developing schizophrenia later in life, providing additional evidence for a link between in utero developmental pathology and risk of developing the condition.<ref name="fn_73">[http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16469941&query_hl=1&itool=pubmed_docsum Brown, A.S. (2006)] Prenatal infection as a risk factor for schizophrenia. ''Schizophrenia Bulletin'', 32 (2), 200-2.<br></ref>
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There is now significant evidence that [[prenatal]] exposure to infections increases the risk for developing [[schizophrenia]] later in life, providing additional evidence for a link between in utero developmental pathology and risk of developing the condition.<ref name="fn_73">[http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16469941&query_hl=1&itool=pubmed_docsum Brown, A.S. (2006)] Prenatal infection as a risk factor for schizophrenia. ''Schizophrenia Bulletin'', 32 (2), 200-2.<br></ref>
   
   

Latest revision as of 14:15, February 22, 2007

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There is now significant evidence that prenatal exposure to infections increases the risk for developing schizophrenia later in life, providing additional evidence for a link between in utero developmental pathology and risk of developing the condition.[1]


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  1. ↑ Brown, A.S. (2006) Prenatal infection as a risk factor for schizophrenia. Schizophrenia Bulletin, 32 (2), 200-2.

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