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{{ClinPsy}}
 
{{ClinPsy}}
The '''biopsychosocial model''' is a general model or approach that posits that biological, psychological (which entails thoughts, emotions, and behaviors), and social factors (abbreviated "BPS") all play a significant role in human functioning in the context of disease or illness. This is in contrast to the traditional, reductionist [[biomedical model]] of medicine that suggests every disease process can be reduced down (i.e., explained) by an underlying process, such as an antigen. <ref>Engel, George L. "The need for a new medical model" 196:129–136, 1977 </ref> The concept is used in fields such as [[medicine]], [[nursing]], [[health psychology]] and [[sociology]], and particularly in more specialist fields such as [[psychiatry]], [[health psychology]] [[chiropractic]] and [[clinical psychology]]. The biopsychosocial paradigm is also a technical term for the popular concept of the mind-body connection, which addresses more philosophical arguments between the biopsychosocial and biomedical models, rather than their empirical exploration and clinical application.<ref>Sarno, John E. MD "The Mindbody Prescription: Healing the Body, Healing the Pain." 1998 [http://www.amazon.com/gp/product/0446675156].</ref>
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The '''biopsychosocial model''' (abbreviated "BPS") is a general model or approach positing that [[biology|biological]], [[psychology|psychological]] (which entails thoughts, emotions, and behaviors), and [[social]] factors, all play a significant role in human functioning in the context of [[disease]] or illness. Indeed, health is best understood in terms of a combination of biological, psychological, and social factors rather than purely in biological terms.<ref name="Santrock, J. W. 2007">Santrock, J. W. (2007). ''A Topical Approach to Human Life-span Development'' (3rd ed.). St. Louis, MO: McGraw-Hill.</ref> This is in contrast to the traditional, [[reductionism|reductionist]] [[biomedical model]] of medicine that suggests every disease process can be explained in terms of an underlying deviation from normal function such as a [[pathogen]], genetic or [[developmental abnormality]], or injury.<ref name=engel77>Engel, George L. (1977). [http://dx.doi.org/10.1126/science.847460 "The need for a new medical model: A challenge for biomedicine".] ''[[Science (journal)|Science]]'' 196:129–136. ISSN 0036-8075 (print) / ISSN
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1095-9203 (web) {{doi|10.1126/science.847460}}</ref> The concept is used in fields such as [[medicine]], [[nursing]], [[health psychology]] and [[sociology]], and particularly in more specialist fields such as [[psychiatry]], [[health psychology]], [[family therapy]], [[chiropractic]], [[clinical social work]], and [[clinical psychology]]. The biopsychosocial paradigm is also a technical term for the popular concept of the "mind–body connection", which addresses more philosophical arguments between the biopsychosocial and biomedical models, rather than their empirical exploration and clinical application.<ref>Sarno, John E. MD "The Mindbody Prescription: Healing the Body, Healing the Pain." 1998 [http://www.amazon.com/gp/product/0446675156].</ref>
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The model was theorized by [[psychiatrist]] [[George L. Engel]] at the [[University of Rochester]], and putatively discussed in a 1977 article in [[Science (journal)|Science]],<ref name=engel77/> where he posited "the need for a new medical model." He discusses his model in detail in his paper in the American Journal of Psychiatry [Engel GL The clinical application of the biopsychosocial model, American Journal of Psychiatry 1980;137:535-544] where he discusses the fate of a hypothetical patient, a 55 year old Mr Glover who has a second heart attack six months after his first. Engel elegantly indicates that Mr Glover's personality helps to interpret his chest pain, that he is in some degree of denial and that it is only the intervention of his employer that gives him permission to seek help. Whereas reductionistically his heart attack can be understood as a clot in a coronary artery, the wider personal perspective helps to understand that different outcomes may be possible depending on how the person responds to his condition. Subsequently Mr Glover in the emergency room develops a cardiac arrest as a result of an incompetent arterial puncture. Once again systems theory can analyse this event in wider terms than just a cardiac arrhythmia. It sees the event as due to inadequate training and supervision of junior staff in an emergency room. Thus while there may be "no single definitive, irreducible model has been published." <ref name="mac">{{cite journal | author = McLaren N | year = 2002 | title = The myth of the biopsychosocial model | url = | journal = Australian and New Zealand Journal of Psychiatry | volume = 36 | issue = 5| pages = 701–703 }}</ref> Engel's elegant exposition of his model in this paper gives plenty of scope for this broader understanding of clinical practice.
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The novelty, acceptance, and prevalence of the biopsychosocial model varies across cultures.<ref name="Santrock, J. W. 2007" />
   
The "model" was theorised by [[psychiatrist]] George Engel at the [[University of Rochester]], and putatively discussed in a [[1977]] article in [[Science (journal)|Science]]<ref>Engel, George L. "The need for a new medical model" 196:129&ndash;136, 1977. PMID 847460.</ref>, where he posited "the need for a new medical model"; however no single definitive, irreducable model has been published.<ref name="mac">McLaren N (2002) "The myth of the biopsychosocial model". Australian and New Zealand Journal of Psychiatry 36 (5), 701–703</ref> However, the general BPS model has guided formulation and testing of models within each professional field (see below re: health behavior models for an example).
 
   
 
==Model description and application in medicine==
 
==Model description and application in medicine==
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Some thinkers see the biopsychosocial model in terms of causation. On this understanding the biological component of the biopsychosocial model seeks to understand how the cause of the illness stems from the functioning of the individual's body. The psychological component of the biopsychosocial model looks for potential psychological causes for a health problem such as lack of self-control, emotional turmoil, and negative thinking. The social part of the biopsychosocial model investigates how different social factors such as socioeconomic status, culture, poverty, technology, and religion can influence health.<ref name="Santrock, J. W. 2007" /> However a closer reading of Engel's seminal paper in the American Journal of Psychiatry (1980) embeds the biopsychosocial model far more closely into patient care. it is not just about causation but about how any clinical condition (medical, surgical or psychiatric) can either be seen narrowly as just biological or more widely as a condition with psychological and social components, which will impinge on a patient's understanding of her condition and will affect the clinical course of that condition.
   
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Drawing on the systems theory of Weiss and von Bertalanffy, Engel describes the commonsense observation that nature is a "hierarchically arranged continuum with its more complex, larger units superordinate on the less complex smaller units." He represents them schematically either as a vertical stack or as a nest of squares, with the simplest at the centre and the most complex on the outside. He subdivided the vertical stack into two stacks. The first starts with sub-atomic particles and ends with the individual person. The second starts with the person and finishes with the biosphere. The first is an organismic hierarchy, the second a social hierarchy. He then delineates some principles: 1. Each level in the system is relatively autonomous. Thus a cell can be studies just as a cell. 2. Each level depends on the level below. Thus a cell is composed of nuclei, mitochondria and all sorts of other organelles. 3. Each level is a component of a higher system. Thus cells organise together to become tissues, and organs etc. Thus, "in the continuity of natural systems every unit is at the very same time both a whole and a part." To these principles Engel developed we can add further that higher level properties emerge from lower level systems and cannot be predicted from studying the lower level, We can also add the principle of top-down causation namely that higher levels can influence lower levels.
The biopsychosocial model implies treatment of disease processes (e.g., type 2 diabetes, cancer, etc.) requires that the health care team address [[biology|biological]], [[psychology|psychological]] and [[society|social]] influences upon a patient's functioning. In a philosophical sense, the biopsychosocial model states that the workings of the body can affect the mind, and the workings of the mind can affect the body.<ref>Halligan, P.W., & Aylward, M. (Eds.) (2006). "The Power of Belief: Psychosocial influence on illness, disability and medicine". Oxford University Press, UK</ref> This means both a direct interaction between mind and body as well as indirect effects through intermediate factors.
 
   
 
The biopsychosocial model of health is based in part on [[social cognitive theory]]. The biopsychosocial model implies that treatment of disease processes, for example type two [[diabetes]] and [[cancer]], requires that the health care team address [[biology|biological]], [[psychology|psychological]] and [[society|social]] influences upon a patient's functioning. In a philosophical sense, the biopsychosocial model states that the workings of the body can affect the mind, and the workings of the mind can affect the body.<ref>Halligan, P.W., & Aylward, M. (Eds.) (2006). "The Power of Belief: Psychosocial influence on illness, disability and medicine". Oxford University Press, UK</ref> This means both a direct interaction between mind and body as well as indirect effects through intermediate factors.
The biopsychosocial model presumes that it is important to handle the three together as a growing body of empirical literature suggests that patient perceptions of health and threat of disease, as well as barriers in a patient's social or cultural environment, appears to influence the likelihood that a patient will engage in health-promoting or treatment behaviors, such as medication taking, proper diet, engaging in physical activity <ref> DiMatteo, M.R., Haskard, K.B., & Williams, S. L. (2007) Health beliefs, disease severity, and patient adherence: A meta-analysis. ''Medical Care'', 45, 521-528.</ref>
 
   
 
The biopsychosocial model presumes that it is important to handle the three together as a growing body of empirical literature suggests that patient perceptions of health and threat of disease, as well as barriers in a patient's social or cultural environment, appear to influence the likelihood that a patient will engage in health-promoting or treatment behaviors, such as medication taking, proper diet or [[nutrition]], and engaging in physical activity.<ref>{{cite journal | author = DiMatteo M.R., Haskard K.B., Williams S. L. | year = 2007 | title = Health beliefs, disease severity, and patient adherence: A meta-analysis | url = | journal = Medical Care | volume = 45 | issue = | pages = 521–528 }}</ref>
While operating from a BPS framework requires that more information be gathered during a consultation, a growing trend in US healthcare (and already well-established in Europe such as in the U.K. & Germany) includes the integration of professional services through integrated disciplinary teams, to provide better care and address the patient's needs at all three levels <ref> Gatchel, R. J. & Oordt, M. S. (2003) Clinical health psychology and primary care: Practical advice and clinical guidance for successful collaboration. American Psychological Association: Washington, D.C. </ref> As seen, for example in integrated primary care clinics, such as used in the U.K., Germany, U.S. Veteran's Administration, U.S. military, Kaiser Permanente, integrated teams may comprise of physicians, nurses, health psychologists, social workers, and other specialties to address all three aspects of the BPS framework, allowing the physician to focus on predominantly biological mechanisms of the patient's complaints <ref> Gatchel, R. J. & Oordt, M. S. (2003) Clinical health psychology and primary care: Practical advice and clinical guidance for successful collaboration. American Psychological Association: Washington, D.C. </ref> See also <ref>Society of Behavioral Medicine</ref>
 
   
 
While operating from a BPS framework requires that more information be gathered during a consultation, a growing trend in US healthcare (and already well-established in Europe such as in the U.K. & Germany) includes the integration of professional services through integrated disciplinary teams, to provide better care and address the patient's needs at all three levels.<ref name="Gatchel, R. J. 2003">Gatchel, R. J. & Oordt, M. S. (2003) Clinical health psychology and primary care: Practical advice and clinical guidance for successful collaboration. American Psychological Association: Washington, D.C.</ref> As seen, for example in integrated primary care clinics, such as used in the U.K., Germany, U.S. Veteran's Administration, U.S. military, Kaiser Permanente, integrated teams may comprise physicians, nurses, health psychologists, social workers, and other specialties to address all three aspects of the BPS framework, allowing the physician to focus on predominantly biological mechanisms of the patient's complaints<ref name="Gatchel, R. J. 2003" /> See also<ref>Society of Behavioral Medicine</ref>
There are also theories that the state of mind directly affects the [[immune system]], and there are many carefully-planned studies that show this to be the case ([[psychoneuroimmunology]]). Psychosocial factors can cause a biological effect by predisposing the patient to risk factors. An example is that [[clinical depression|depression]] by itself may not cause [[liver]] problems, but a depressed person may be more likely to have [[ethanol|alcohol]] problems, thus liver damage. It is this increased risk-taking that can also lead to an increased likelihood of disease. Most of the diseases referred to in BPS discussion tend to be such behaviourally-moderated illnesses which have known high risk factors, or so-called "biopsychosocial illnesses/disorders" <ref>Bruns D, Disorbio JM, "Chronic Pain and Biopsychosocial Disorders". Practical Pain Management, March 2006, volume 6, issue 2 [http://www.pearsonassessments.com/resources/ppm-1105-bruns.pdf]</ref><ref>[http://www.healthpsych.com/research/research.html An Overview Of Biopsychosocial Disorders: Conceptualization, Assessment And Treatment]</ref> An example of this is type 2 diabetes, which with the growing prevalence of obesity and physical inactivity, is on course to become a worldwide pandemic (e.g., approximately 20 million Americans are estimated to have diabetes, with 90% to 95% considered type 2). <ref> Wild, S., Roglic, G., Green, A., Sicree, R., & King, H. (2004). Global prevalence of diabetes: Estimates for the year 2000 and projections for 2030. Diabetes Care, 27, 1047-1053. </ref>
 
   
 
Psychosocial factors can cause a biological effect by predisposing the patient to risk factors. An example is that [[clinical depression|depression]] by itself may not cause [[liver]] problems, but a depressed person may be more likely to have [[ethanol|alcohol]] problems, and therefore liver damage. Perhaps it is this increased risk-taking that leads to an increased likelihood of disease. Most diseases in BPS discussion are such behaviourally-moderated illnesses, with known high risk factors, or so-called "biopsychosocial illnesses/disorders".<ref>Bruns D, Disorbio JM, "Chronic Pain and Biopsychosocial Disorders". Practical Pain Management, March 2006, volume 6, issue 2 [http://www.pearsonassessments.com/resources/ppm-1105-bruns.pdf]</ref><ref>[http://www.healthpsych.com/research/research.html An Overview Of Biopsychosocial Disorders: Conceptualization, Assessment And Treatment]</ref> An example of this is type 2 diabetes, which with the growing prevalence of obesity and physical inactivity, is on course to become a worldwide pandemic. For example, approximately 20 million Americans are estimated to have diabetes, with 90% to 95% considered type 2.<ref>{{cite journal | author = Wild S., Roglic G., Green A., Sicree R., King H. | year = 2004 | title = Global prevalence of diabetes: Estimates for the year 2000 and projections for 2030 | url = | journal = Diabetes Care | volume = 27 | issue = | pages = 1047–1053 }}</ref>
It is important to note that the biopsychosocial model does not provide a straightforward, testable model to explain the interactions or causal influences (i.e., amount of variance accounted for) by each of the components (biological, psychological, or social). Rather, the model has been a general framework to guide theoretical and empirical exploration, which has amassed a great deal of research since Engel's 1977 article. One of the areas that has been greatly influenced is the formulation and testing of social-cognitive models of health behavior over the past 30 years. <ref> Armitage, C. J., & Conner, M. (2000). Social cognition models and health behaviour: A structured review. Psychology and Health, 15, 173-189. </ref>. While no single model has taken precedence, a large body of empirical literature has identified social-cognitive (the psyho-social aspect of Engel's model) variables that appear to influence engagement in healthy behaviors and adhere to prescribed medical regimens, such as self-efficacy, in chronic diseases such as type 2 diabetes, cardiovascular disease, etc. <ref>Allen, N. A. (2004). Social cognitive theory in diabetes exercise research: An integrative literature review. The Diabetes Educator, 30, 805-819. </ref> <ref>Carlson, J. J., Norman, G. J., Feltz, D. L., Franklin, B. A., Johnson, J. A., & Locke, S. K. (2001). Self-efficacy, psychosocial factors, and exercise behavior in traditional verses modified cardiac rehabilitation. Journal of Cardiopulmonary Rehabilitation. 21, 363-373. </ref> These models include the Health Belief Model, Theory of Reasoned Action and Theory of Planned Behavior, Transtheortical Model, the Relapse Prevention Model, Gollwitzer's implementation-intentions, the Precaution-Adoption Model, the Health Action Process Approach, etc... <ref>Armitage, C. J., & Conner, M. (2000). Social cognition models and health behaviour: A structured review. Psychology and Health, 15, 173-189.</ref><ref>Schwarzer, R. (1992). Self-efficacy in the adoption and maintenance of health behaviors: theoretical approaches and a new model. In R. Schwarzer (Ed.), Self-efficacy: Thought control of action. London, England: Hemisphere Publishing Corporation.</ref><ref> Garcia, K. & Mann, T. (2003). Form ‘I wish’ to ‘I will’: Social-cognitive predictors of behavioral intentions. Journal of Health Psychology, 8, 347-360.</ref><ref>Carels, R. A., Douglass, O. M., Cacciapaglia, H. M., & O’Brien, W. H. (2004). An ecological momentary assessment of relapse crises in dieting. Journal of Consulting and Clinical Psychology, 72, 341-348. </ref><ref>Carels, R. A., Darby, L. A. Rydin, S., Douglass, O. M., Cacciapaglia, H. M., & O’Brien, W. H. (2005). The relationship between self-monitoring, outcome expectancies, difficulties with eating and exercise, and physical activity and weight loss treatment outcomes. Annals of Behavioral Medicine. 30(3), 182-190. </ref><ref> Blanchard, C. M., Courneya, K. S., Rodgers, W. M., Frasier, S. N., Murray, T., Daub, B., & Black, B. (2003). Is the theory of planned behavior a useful framework for understanding exercise adherence during phase II cardiac rehabilitation? Journal of Cardiopulmonary Rehabilitation, 23, 29-39. </ref>
 
   
 
It is important to note that the biopsychosocial model does not provide a straightforward, testable model to explain the interactions or causal influences (that is, amount of variance accounted for) by each of the components (biological, psychological, or social). Rather, the model has been a general framework to guide theoretical and empirical exploration, which has amassed a great deal of research since Engel's 1977 article. One of the areas that has been greatly influenced is the formulation and testing of social-cognitive models of health behavior over the past 30 years.<ref name="Armitage, C. J. 2000">{{cite journal | author = Armitage C. J., Conner M. | year = 2000 | title = Social cognition models and health behaviour: A structured review | url = | journal = Psychology and Health | volume = 15 | issue = | pages = 173–189 }}</ref> While no single model has taken precedence, a large body of empirical literature has identified social-cognitive (the psycho-social aspect of Engel's model) variables that appear to influence engagement in healthy behaviors and adhere to prescribed medical regimens, such as self-efficacy, in chronic diseases such as type 2 diabetes, cardiovascular disease, etc.<ref>{{cite journal | author = Allen N. A. | year = 2004 | title = Social cognitive theory in diabetes exercise research: An integrative literature review | url = | journal = The Diabetes Educator | volume = 30 | issue = | pages = 805–819 }}</ref><ref>{{cite journal | author = Carlson J. J., Norman G. J., Feltz D. L., Franklin B. A., Johnson J. A., Locke S. K. | year = 2001 | title = Self-efficacy, psychosocial factors, and exercise behavior in traditional verses modified cardiac rehabilitation | url = | journal = Journal of Cardiopulmonary Rehabilitation | volume = 21 | issue = | pages = 363–373 }}</ref> These models include the [[Health Belief Model]], [[Theory of Reasoned Action]] and [[Theory of Planned Behavior]], [[Transtheoretical Model]], the Relapse Prevention Model, Gollwitzer's [[Implementation intention|implementation-intentions]], the Precaution–Adoption Model, the [[Health Action Process Approach]], etc.<ref name="Armitage, C. J. 2000" /><ref>Schwarzer, R. (1992). Self-efficacy in the adoption and maintenance of health behaviors: theoretical approaches and a new model. In R. Schwarzer (Ed.), Self-efficacy: Thought control of action. London, England: Hemisphere Publishing Corporation.</ref><ref>{{cite journal | author = Garcia K., Mann T. | year = 2003 | title = From 'I wish' to 'I will': Social-cognitive predictors of behavioral intentions | url = | journal = [[Journal of Health Psychology]] | volume = 8 | issue = | pages = 347–360 }}</ref><ref>{{cite journal | author = Carels R. A., Douglass O. M., Cacciapaglia H. M., O'Brien W. H. | year = 2004 | title = An ecological momentary assessment of relapse crises in dieting | url = | journal = Journal of Consulting and Clinical Psychology | volume = 72 | issue = | pages = 341–348 }}</ref><ref>{{cite journal | author = Carels R. A., Darby L. A. Rydin, Douglass O. M., Cacciapaglia H. M., O'Brien W. H. | year = 2005 | title = The relationship between self-monitoring, outcome expectancies, difficulties with eating and exercise, and physical activity and weight loss treatment outcomes | url = | journal = Annals of Behavioral Medicine | volume = 30 | issue = 3| pages = 182–190 }}</ref><ref>{{cite journal | author = Blanchard C. M., Courneya K. S., Rodgers W. M., Frasier S. N., Murray T., Daub B., Black B. | year = 2003 | title = Is the theory of planned behavior a useful framework for understanding exercise adherence during phase II cardiac rehabilitation? | url = | journal = Journal of Cardiopulmonary Rehabilitation | volume = 23 | issue = | pages = 29–39 }}</ref>
==Criticism of the Biopsychosocial model==
 
   
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==Criticism==
Some critics point out this question of distinction and of determination of the roles of illness and disease runs against the growing concept of the patient-doctor partnership or [[patient empowerment]], as "biopsychosocial" becomes one more disengenous [[euphemism]] for [[psychosomatic]] illness.<ref>McLaren N. "The Biopsychosocial Model and Scientific Fraud". {Paper presented to RANZCP Congress, Christchurch NZ May 2004. Revised version: "When does Self-Deception become Culpable?" Chap.8 in McLaren N. "Humanizing Madness: Psychiatry and the cognitive neurosciences" ISBN 9781932690392</ref> This may be exploitated by medical insurance companies or government welfare departments eager to limit or deny access to medical and social care<ref>Rutherford J. [http://www.compassonline.org.uk/article.asp?n=563&offset=20 New Labour and the end of welfare] Compass Online April 25 2007</ref>.
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Some critics point out this question of distinction and a question of determination of the roles of illness and disease runs against the growing concept of the patient–medical tradesperson partnership or [[patient empowerment]], as "biopsychosocial" becomes one more disingenuous [[euphemism]] for [[psychosomatic]] illness.<ref>McLaren N. "The Biopsychosocial Model and Scientific Fraud". {Paper presented to RANZCP Congress, Christchurch NZ May 2004. Revised version: "When does Self-Deception become Culpable?" Chap.8 in McLaren N. "Humanizing Madness: Psychiatry and the cognitive neurosciences" ISBN 978-1-932690-39-2</ref> This may be exploited by medical insurance companies or government welfare departments eager to limit or deny access to medical and social care.<ref>Rutherford J. [http://www.compassonline.org.uk/news/item.asp?n=563&offset=20 New Labour and the end of welfare] Compass Online April 25, 2007</ref>
   
Some psychiatrists see the BPS model as flawed, in either formulation or application. Epstein et al describes six conflicting interpretations of what the model might be, and proposes that "...habits of mind may be the missing link between a biopsychosocial intent and clinical reality."<ref>Epstein RM, Borrell-Carrio F, "The biopsychosocial model: exploring six impossible things". Families, Systems & Health 22 Dec 2005</ref> Rather than the result of the BPS model, David Pilgrim suggests that neccessitous pragmatism and a form of "mutual tolerance" (Goldie, 1977) has forced a co-existence and not "genuine evidence of theoretical integration as a shared BPS orthodoxy."<ref name="pilg">Pilgrim D. "The biopsychosocial model in Anglo-American psychiatry: Past, present and future" Journal of Mental Health, Volume 11, Issue 6 December 2002 , pages 585 - 594 DOI 10.1080/09638230020023930</ref> Pilgrim goes on to state, "Despite these scientific and ethical virtues," the BPS model "...has not been properly realised. It seems to have been pushed into the shadows by a return to medicine and the re-ascendancy of a biomedical model." <ref>The biopsychosocial model in Anglo-American psychiatry: Past, present and future" Journal of Mental Health, Volume 11, Issue 6 December 2002 , pages 585 - 594 DOI 10.1080/09638230020023930</ref>
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Some psychiatrists see the BPS model as flawed, in either formulation or application. Epstein and colleagues describe six conflicting interpretations of what the model might be, and proposes that "...habits of mind may be the missing link between a biopsychosocial intent and clinical reality."<ref>Epstein RM, Borrell-Carrio F, "The biopsychosocial model: exploring six impossible things". Families, Systems & Health 22 Dec 2005</ref> Psychiatrist Hamid Tavakoli argues that the BPS model should be avoided because it unintentionally promotes an artificial distinction between biology and psychology, and merely causes confusion in psychiatric assessments and training programs, and that ultimately it has not helped the cause of trying to destigmatize mental health.<ref>{{cite journal | pmc = 2719450 | title = A Closer Evaluation of Current Methods in Psychiatric Assessments: A Challenge for the Biopsychosocial Model | author= Hamid R. Tavakoli, MD | pmid=19724745 | volume=6 | issue=2 | year=2009 | month=February | journal=Psychiatry (Edgmont) | pages=25–30}}</ref>
   
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Sociologist David Pilgrim suggests that a necessary pragmatism and a form of "mutual tolerance" (Goldie, 1977) has forced a co-existence of perspectives, rather than a genuine "theoretical integration as a shared BPS orthodoxy."<ref name="pilg">Pilgrim D. "The biopsychosocial model in Anglo-American psychiatry: Past, present and future" ''Journal of Mental Health'', Volume 11, Issue 6 December 2002 , pages 585 - 594 {{doi|10.1080/09638230020023930}}</ref> Pilgrim goes on to state that despite "scientific and ethical virtues," the BPS model "...has not been properly realised. It seems to have been pushed into the shadows by a return to medicine and the re-ascendancy of a biomedical model."<ref>The biopsychosocial model in Anglo-American psychiatry: Past, present and future" ''Journal of Mental Health'', Volume 11, Issue 6, pages 585 - 594 {{doi|10.1080/09638230020023930}}</ref>
Perhaps the most vocal philosophical critic of the BPS model, Niall McLaren (ISBN 9781932690392) writes:
 
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However, a vocal philosophical critic of the BPS model, psychiatrist Niall McLaren,<ref name="McLaren 2007">{{Cite book
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| title = Humanizing Madness
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| last = McLaren | first = Niall
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| year = 2007
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| isbn = 1-932690-39-5
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| pages =
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| publisher = Loving Healing Press
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| location = Ann Arbor, MI}}{{Page needed|date=September 2010}}</ref> writes:
 
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<blockquote>
"Since the collapse of the 19th century models (psychoanalysis, biologism and behaviourism), psychiatrists have been in search of a model which integrates the psyche and the soma. So keen has been their search that they embraced the so-called ‘biopsychosocial model’ without ever bothering to check its details. If, at any time over the last three decades, they had done so, they would have found it had none. This would have forced them into the embarrassing position of having to acknowledge that modern psychiatry is operating in a theoretical vacuum."<ref>McLaren N. "Interactive dualism as a partial solution to the mind-brain problem for psychiatry". Med Hypotheses 2006;66(6):1165-73</ref></blockquote>
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"Since the collapse of the 19th century models (psychoanalysis, biologism and behaviourism), psychiatrists have been in search of a model that integrates the psyche and the soma. So keen has been their search that they embraced the so-called 'biopsychosocial model' without ever bothering to check its details. If, at any time over the last three decades, they had done so, they would have found it had none. This would have forced them into the embarrassing position of having to acknowledge that modern psychiatry is operating in a theoretical vacuum."<ref>{{cite journal | author = McLaren N | year = 2006 | title = Interactive dualism as a partial solution to the mind-brain problem for psychiatry | url = | journal = Med Hypotheses | volume = 66 | issue = 6| pages = 1165–73 }}</ref></blockquote>
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The rationale for this theoretical vacuum is outlined in his 1998 paper<ref name="McLaren N 1998 86–92; discussion 93–6">{{Cite journal|author=McLaren N |title=A critical review of the biopsychosocial model |journal=The Australian and New Zealand Journal of Psychiatry |volume=32 |issue=1 |pages=86–92; discussion 93–6 |year=1998 |month=February |pmid=9565189 |doi=10.1046/j.1440-1614.1998.00343.x}}</ref> and more recently in his books, most notably ''Humanizing Psychiatrists''.<ref name=McLaren2010>{{Cite book
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| title = Humanizing Psychiatrists
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| last = McLaren | first = Niall
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| year = 2010
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| isbn = 978-1-61599-060-3
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| pages = 135–154
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| publisher = Loving Healing Press
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| location = Ann Arbor, MI}}</ref> Simply put, the purpose of a [[scientific model]] is to see if a [[scientific theory]] works and to actualize its logical consequences. In this sense, models are real and their material consequences can be measured, whereas theories are ideas and can no more be measured than daydreams. Model-building separates theories with a future from those that always remain dreams. An example of a true scientific model is longer necked giraffes reach more food, survive at higher rates, and pass on this longer neck trait to their progeny. This is a model (natural selection) of the theory of evolution. Therefore, from an [[epistemology|epistemological]] stance there can be no model of mental disorder without first establishing a [[theory of mind|theory of the mind]]. Dr. McLaren does not say that the biopsychosocial model is devoid of merit, just that it does not fit the definition of a scientific model (or theory) and does not "reveal anything that would not be known (implicitly, if not explicitly) to any practitioner of reasonable sensitivity." He states that the biopsychosocial model should be seen in a historical context as bucking against the trend of [[biological reductionism]], which was (and still is) overtaking psychiatry. Engel "has done a very great service to orthodox psychiatry in that he legitimised the concept of talking to people as people." In short, even though it is correct to say that sociology, psychology, and biology are factors in mental illness, simply stating this obvious fact does not make it a model in the scientific sense of the word.<ref name="McLaren 2007"/><ref name="McLaren N 1998 86–92; discussion 93–6"/><ref name=McLaren2010/><ref>{{Cite book
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| title = Humanizing Psychiatry
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| last = McLaren | first = Niall
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| year = 2009
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| isbn = 1-61599-011-9
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| pages =
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| publisher = Loving Healing Press
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| location = Ann Arbor, MI}}{{Page needed|date=September 2010}}</ref>
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The psychiatry professor and author [[S. Nassir Ghaemi]] considers Engel's model to be anti-humanistic and advocates the use of less eclectic, less generic and less vague alternatives such as [[William Osler]]’s medical humanism or [[Karl Jaspers]]’ method-based psychiatry. <ref> Ghaemi S.N. (2009) ''The rise and fall of the biopsychosocial model'' The British Journal of Psychiatry 195: 3-4 {{doi|10.1192/bjp.bp.109.063859}} </ref>
   
McLaren claims that his model of emergent dualist interaction, based in the work of Alan Turing and David Chalmers, more than satisfies the expectations institutional psychiatry has placed upon Engel's ambitious "model."
 
   
 
==See also==
 
==See also==
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* [[Biosocial perspective in health psychology]]
 
* [[Biosocial perspective in health psychology]]
 
* [[Body-mind]]
 
* [[Body-mind]]
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* [[Diathesis–stress model]]
 
* [[Health psychology]]
 
* [[Health psychology]]
 
* [[Holistic health]]
 
* [[Holistic health]]
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== References ==
 
== References ==
 
{{reflist}}
 
{{reflist}}
  +
  +
==Further reading==
  +
*Melchert, Timothy P.. Foundations of professional psychology: the end of theoretical orientations and the emergence of the biopsychosocial approach. London: Elsevier, 2011.
   
 
==External links==
 
==External links==
 
 
* Bracken, Patrick, Thomas, Philip, [http://www.bmj.com/cgi/content/full/325/7378/1433 "Time to move beyond the mind-body split"], editorial, [[British Medical Journal]] 2002;325:1433-1434 (21 December)
 
* Bracken, Patrick, Thomas, Philip, [http://www.bmj.com/cgi/content/full/325/7378/1433 "Time to move beyond the mind-body split"], editorial, [[British Medical Journal]] 2002;325:1433-1434 (21 December)
   
   
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[[Category:Biopsychosocial approach]]
 
 
[[Category:Medical sociology]]
 
[[Category:Medical sociology]]
 
[[Category:Specific models]]
 
[[Category:Specific models]]

Revision as of 22:37, 8 August 2013

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The biopsychosocial model (abbreviated "BPS") is a general model or approach positing that biological, psychological (which entails thoughts, emotions, and behaviors), and social factors, all play a significant role in human functioning in the context of disease or illness. Indeed, health is best understood in terms of a combination of biological, psychological, and social factors rather than purely in biological terms.[1] This is in contrast to the traditional, reductionist biomedical model of medicine that suggests every disease process can be explained in terms of an underlying deviation from normal function such as a pathogen, genetic or developmental abnormality, or injury.[2] The concept is used in fields such as medicine, nursing, health psychology and sociology, and particularly in more specialist fields such as psychiatry, health psychology, family therapy, chiropractic, clinical social work, and clinical psychology. The biopsychosocial paradigm is also a technical term for the popular concept of the "mind–body connection", which addresses more philosophical arguments between the biopsychosocial and biomedical models, rather than their empirical exploration and clinical application.[3]

The model was theorized by psychiatrist George L. Engel at the University of Rochester, and putatively discussed in a 1977 article in Science,[2] where he posited "the need for a new medical model." He discusses his model in detail in his paper in the American Journal of Psychiatry [Engel GL The clinical application of the biopsychosocial model, American Journal of Psychiatry 1980;137:535-544] where he discusses the fate of a hypothetical patient, a 55 year old Mr Glover who has a second heart attack six months after his first. Engel elegantly indicates that Mr Glover's personality helps to interpret his chest pain, that he is in some degree of denial and that it is only the intervention of his employer that gives him permission to seek help. Whereas reductionistically his heart attack can be understood as a clot in a coronary artery, the wider personal perspective helps to understand that different outcomes may be possible depending on how the person responds to his condition. Subsequently Mr Glover in the emergency room develops a cardiac arrest as a result of an incompetent arterial puncture. Once again systems theory can analyse this event in wider terms than just a cardiac arrhythmia. It sees the event as due to inadequate training and supervision of junior staff in an emergency room. Thus while there may be "no single definitive, irreducible model has been published." [4] Engel's elegant exposition of his model in this paper gives plenty of scope for this broader understanding of clinical practice.

The novelty, acceptance, and prevalence of the biopsychosocial model varies across cultures.[1]


Model description and application in medicine

Some thinkers see the biopsychosocial model in terms of causation. On this understanding the biological component of the biopsychosocial model seeks to understand how the cause of the illness stems from the functioning of the individual's body. The psychological component of the biopsychosocial model looks for potential psychological causes for a health problem such as lack of self-control, emotional turmoil, and negative thinking. The social part of the biopsychosocial model investigates how different social factors such as socioeconomic status, culture, poverty, technology, and religion can influence health.[1] However a closer reading of Engel's seminal paper in the American Journal of Psychiatry (1980) embeds the biopsychosocial model far more closely into patient care. it is not just about causation but about how any clinical condition (medical, surgical or psychiatric) can either be seen narrowly as just biological or more widely as a condition with psychological and social components, which will impinge on a patient's understanding of her condition and will affect the clinical course of that condition.

Drawing on the systems theory of Weiss and von Bertalanffy, Engel describes the commonsense observation that nature is a "hierarchically arranged continuum with its more complex, larger units superordinate on the less complex smaller units." He represents them schematically either as a vertical stack or as a nest of squares, with the simplest at the centre and the most complex on the outside. He subdivided the vertical stack into two stacks. The first starts with sub-atomic particles and ends with the individual person. The second starts with the person and finishes with the biosphere. The first is an organismic hierarchy, the second a social hierarchy. He then delineates some principles: 1. Each level in the system is relatively autonomous. Thus a cell can be studies just as a cell. 2. Each level depends on the level below. Thus a cell is composed of nuclei, mitochondria and all sorts of other organelles. 3. Each level is a component of a higher system. Thus cells organise together to become tissues, and organs etc. Thus, "in the continuity of natural systems every unit is at the very same time both a whole and a part." To these principles Engel developed we can add further that higher level properties emerge from lower level systems and cannot be predicted from studying the lower level, We can also add the principle of top-down causation namely that higher levels can influence lower levels.

The biopsychosocial model of health is based in part on social cognitive theory. The biopsychosocial model implies that treatment of disease processes, for example type two diabetes and cancer, requires that the health care team address biological, psychological and social influences upon a patient's functioning. In a philosophical sense, the biopsychosocial model states that the workings of the body can affect the mind, and the workings of the mind can affect the body.[5] This means both a direct interaction between mind and body as well as indirect effects through intermediate factors.

The biopsychosocial model presumes that it is important to handle the three together as a growing body of empirical literature suggests that patient perceptions of health and threat of disease, as well as barriers in a patient's social or cultural environment, appear to influence the likelihood that a patient will engage in health-promoting or treatment behaviors, such as medication taking, proper diet or nutrition, and engaging in physical activity.[6]

While operating from a BPS framework requires that more information be gathered during a consultation, a growing trend in US healthcare (and already well-established in Europe such as in the U.K. & Germany) includes the integration of professional services through integrated disciplinary teams, to provide better care and address the patient's needs at all three levels.[7] As seen, for example in integrated primary care clinics, such as used in the U.K., Germany, U.S. Veteran's Administration, U.S. military, Kaiser Permanente, integrated teams may comprise physicians, nurses, health psychologists, social workers, and other specialties to address all three aspects of the BPS framework, allowing the physician to focus on predominantly biological mechanisms of the patient's complaints[7] See also[8]

Psychosocial factors can cause a biological effect by predisposing the patient to risk factors. An example is that depression by itself may not cause liver problems, but a depressed person may be more likely to have alcohol problems, and therefore liver damage. Perhaps it is this increased risk-taking that leads to an increased likelihood of disease. Most diseases in BPS discussion are such behaviourally-moderated illnesses, with known high risk factors, or so-called "biopsychosocial illnesses/disorders".[9][10] An example of this is type 2 diabetes, which with the growing prevalence of obesity and physical inactivity, is on course to become a worldwide pandemic. For example, approximately 20 million Americans are estimated to have diabetes, with 90% to 95% considered type 2.[11]

It is important to note that the biopsychosocial model does not provide a straightforward, testable model to explain the interactions or causal influences (that is, amount of variance accounted for) by each of the components (biological, psychological, or social). Rather, the model has been a general framework to guide theoretical and empirical exploration, which has amassed a great deal of research since Engel's 1977 article. One of the areas that has been greatly influenced is the formulation and testing of social-cognitive models of health behavior over the past 30 years.[12] While no single model has taken precedence, a large body of empirical literature has identified social-cognitive (the psycho-social aspect of Engel's model) variables that appear to influence engagement in healthy behaviors and adhere to prescribed medical regimens, such as self-efficacy, in chronic diseases such as type 2 diabetes, cardiovascular disease, etc.[13][14] These models include the Health Belief Model, Theory of Reasoned Action and Theory of Planned Behavior, Transtheoretical Model, the Relapse Prevention Model, Gollwitzer's implementation-intentions, the Precaution–Adoption Model, the Health Action Process Approach, etc.[12][15][16][17][18][19]

Criticism

Some critics point out this question of distinction and a question of determination of the roles of illness and disease runs against the growing concept of the patient–medical tradesperson partnership or patient empowerment, as "biopsychosocial" becomes one more disingenuous euphemism for psychosomatic illness.[20] This may be exploited by medical insurance companies or government welfare departments eager to limit or deny access to medical and social care.[21]

Some psychiatrists see the BPS model as flawed, in either formulation or application. Epstein and colleagues describe six conflicting interpretations of what the model might be, and proposes that "...habits of mind may be the missing link between a biopsychosocial intent and clinical reality."[22] Psychiatrist Hamid Tavakoli argues that the BPS model should be avoided because it unintentionally promotes an artificial distinction between biology and psychology, and merely causes confusion in psychiatric assessments and training programs, and that ultimately it has not helped the cause of trying to destigmatize mental health.[23]

Sociologist David Pilgrim suggests that a necessary pragmatism and a form of "mutual tolerance" (Goldie, 1977) has forced a co-existence of perspectives, rather than a genuine "theoretical integration as a shared BPS orthodoxy."[24] Pilgrim goes on to state that despite "scientific and ethical virtues," the BPS model "...has not been properly realised. It seems to have been pushed into the shadows by a return to medicine and the re-ascendancy of a biomedical model."[25]

However, a vocal philosophical critic of the BPS model, psychiatrist Niall McLaren,[26] writes:

"Since the collapse of the 19th century models (psychoanalysis, biologism and behaviourism), psychiatrists have been in search of a model that integrates the psyche and the soma. So keen has been their search that they embraced the so-called 'biopsychosocial model' without ever bothering to check its details. If, at any time over the last three decades, they had done so, they would have found it had none. This would have forced them into the embarrassing position of having to acknowledge that modern psychiatry is operating in a theoretical vacuum."[27]

The rationale for this theoretical vacuum is outlined in his 1998 paper[28] and more recently in his books, most notably Humanizing Psychiatrists.[29] Simply put, the purpose of a scientific model is to see if a scientific theory works and to actualize its logical consequences. In this sense, models are real and their material consequences can be measured, whereas theories are ideas and can no more be measured than daydreams. Model-building separates theories with a future from those that always remain dreams. An example of a true scientific model is longer necked giraffes reach more food, survive at higher rates, and pass on this longer neck trait to their progeny. This is a model (natural selection) of the theory of evolution. Therefore, from an epistemological stance there can be no model of mental disorder without first establishing a theory of the mind. Dr. McLaren does not say that the biopsychosocial model is devoid of merit, just that it does not fit the definition of a scientific model (or theory) and does not "reveal anything that would not be known (implicitly, if not explicitly) to any practitioner of reasonable sensitivity." He states that the biopsychosocial model should be seen in a historical context as bucking against the trend of biological reductionism, which was (and still is) overtaking psychiatry. Engel "has done a very great service to orthodox psychiatry in that he legitimised the concept of talking to people as people." In short, even though it is correct to say that sociology, psychology, and biology are factors in mental illness, simply stating this obvious fact does not make it a model in the scientific sense of the word.[26][28][29][30]

The psychiatry professor and author S. Nassir Ghaemi considers Engel's model to be anti-humanistic and advocates the use of less eclectic, less generic and less vague alternatives such as William Osler’s medical humanism or Karl Jaspers’ method-based psychiatry. [31]


See also

References

  1. 1.0 1.1 1.2 Santrock, J. W. (2007). A Topical Approach to Human Life-span Development (3rd ed.). St. Louis, MO: McGraw-Hill.
  2. 2.0 2.1 Engel, George L. (1977). "The need for a new medical model: A challenge for biomedicine". Science 196:129–136. ISSN 0036-8075 (print) / ISSN 1095-9203 (web) DOI:10.1126/science.847460
  3. Sarno, John E. MD "The Mindbody Prescription: Healing the Body, Healing the Pain." 1998 [1].
  4. McLaren N (2002). The myth of the biopsychosocial model. Australian and New Zealand Journal of Psychiatry 36 (5): 701–703.
  5. Halligan, P.W., & Aylward, M. (Eds.) (2006). "The Power of Belief: Psychosocial influence on illness, disability and medicine". Oxford University Press, UK
  6. DiMatteo M.R., Haskard K.B., Williams S. L. (2007). Health beliefs, disease severity, and patient adherence: A meta-analysis. Medical Care 45: 521–528.
  7. 7.0 7.1 Gatchel, R. J. & Oordt, M. S. (2003) Clinical health psychology and primary care: Practical advice and clinical guidance for successful collaboration. American Psychological Association: Washington, D.C.
  8. Society of Behavioral Medicine
  9. Bruns D, Disorbio JM, "Chronic Pain and Biopsychosocial Disorders". Practical Pain Management, March 2006, volume 6, issue 2 [2]
  10. An Overview Of Biopsychosocial Disorders: Conceptualization, Assessment And Treatment
  11. Wild S., Roglic G., Green A., Sicree R., King H. (2004). Global prevalence of diabetes: Estimates for the year 2000 and projections for 2030. Diabetes Care 27: 1047–1053.
  12. 12.0 12.1 Armitage C. J., Conner M. (2000). Social cognition models and health behaviour: A structured review. Psychology and Health 15: 173–189.
  13. Allen N. A. (2004). Social cognitive theory in diabetes exercise research: An integrative literature review. The Diabetes Educator 30: 805–819.
  14. Carlson J. J., Norman G. J., Feltz D. L., Franklin B. A., Johnson J. A., Locke S. K. (2001). Self-efficacy, psychosocial factors, and exercise behavior in traditional verses modified cardiac rehabilitation. Journal of Cardiopulmonary Rehabilitation 21: 363–373.
  15. Schwarzer, R. (1992). Self-efficacy in the adoption and maintenance of health behaviors: theoretical approaches and a new model. In R. Schwarzer (Ed.), Self-efficacy: Thought control of action. London, England: Hemisphere Publishing Corporation.
  16. Garcia K., Mann T. (2003). From 'I wish' to 'I will': Social-cognitive predictors of behavioral intentions. Journal of Health Psychology 8: 347–360.
  17. Carels R. A., Douglass O. M., Cacciapaglia H. M., O'Brien W. H. (2004). An ecological momentary assessment of relapse crises in dieting. Journal of Consulting and Clinical Psychology 72: 341–348.
  18. Carels R. A., Darby L. A. Rydin, Douglass O. M., Cacciapaglia H. M., O'Brien W. H. (2005). The relationship between self-monitoring, outcome expectancies, difficulties with eating and exercise, and physical activity and weight loss treatment outcomes. Annals of Behavioral Medicine 30 (3): 182–190.
  19. Blanchard C. M., Courneya K. S., Rodgers W. M., Frasier S. N., Murray T., Daub B., Black B. (2003). Is the theory of planned behavior a useful framework for understanding exercise adherence during phase II cardiac rehabilitation?. Journal of Cardiopulmonary Rehabilitation 23: 29–39.
  20. McLaren N. "The Biopsychosocial Model and Scientific Fraud". {Paper presented to RANZCP Congress, Christchurch NZ May 2004. Revised version: "When does Self-Deception become Culpable?" Chap.8 in McLaren N. "Humanizing Madness: Psychiatry and the cognitive neurosciences" ISBN 978-1-932690-39-2
  21. Rutherford J. New Labour and the end of welfare Compass Online April 25, 2007
  22. Epstein RM, Borrell-Carrio F, "The biopsychosocial model: exploring six impossible things". Families, Systems & Health 22 Dec 2005
  23. Hamid R. Tavakoli, MD (February 2009). A Closer Evaluation of Current Methods in Psychiatric Assessments: A Challenge for the Biopsychosocial Model. Psychiatry (Edgmont) 6 (2): 25–30.
  24. Pilgrim D. "The biopsychosocial model in Anglo-American psychiatry: Past, present and future" Journal of Mental Health, Volume 11, Issue 6 December 2002 , pages 585 - 594 DOI:10.1080/09638230020023930
  25. The biopsychosocial model in Anglo-American psychiatry: Past, present and future" Journal of Mental Health, Volume 11, Issue 6, pages 585 - 594 DOI:10.1080/09638230020023930
  26. 26.0 26.1 McLaren, Niall (2007). Humanizing Madness, Ann Arbor, MI: Loving Healing Press.Template:Page needed
  27. McLaren N (2006). Interactive dualism as a partial solution to the mind-brain problem for psychiatry. Med Hypotheses 66 (6): 1165–73.
  28. 28.0 28.1 McLaren N (February 1998). A critical review of the biopsychosocial model. The Australian and New Zealand Journal of Psychiatry 32 (1): 86–92; discussion 93–6.
  29. 29.0 29.1 McLaren, Niall (2010). Humanizing Psychiatrists, 135–154, Ann Arbor, MI: Loving Healing Press.
  30. McLaren, Niall (2009). Humanizing Psychiatry, Ann Arbor, MI: Loving Healing Press.Template:Page needed
  31. Ghaemi S.N. (2009) The rise and fall of the biopsychosocial model The British Journal of Psychiatry 195: 3-4 DOI:10.1192/bjp.bp.109.063859

Further reading

  • Melchert, Timothy P.. Foundations of professional psychology: the end of theoretical orientations and the emergence of the biopsychosocial approach. London: Elsevier, 2011.

External links

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